Is this personal experience talking? I'm not totally dismissing the idea since I can see how low doses might work, but I'd be concerned about it being sustainable since I don't know anyone who's tried that for any length of time.
Atomoxetine (Strattera) achieves comparable cortical effects by inhibiting the "norepinephrine" transporter in prefrontal cortex. (Scare quotes because it's also the primary dopamine uptake mechanism in PFC.)
It also has way more fucked up side effects, and this minor corner of about 25% that go into a near psychotic phase for the first month. Of course, thats not to say Adderall is utter shit for adults to take compared to pretty much the majority of alternatives (since how it metabolizes compared to something like Dexedrine, as it's simply really made for a child's metabolism, but I don't think we should give speed to kids), including many that have taken off the market.
Incidentally he really does have ADD, and he went on his own program and he is visibly better - he used to wander off onto unrelated topics constantly and he's quit doing it.
"Experts agree that ADD is multicausal. That is, in any one case there are usually several factors that contribute to the disease. These are: nutritional deficiencies, food allergies, chemical sensitivities, and toxic exposures (from food, air, water, chemicals, and drugs)."
And certainly nothing to do with all these dopamine/norepinephrine polymorphisms we keep finding they have, or the fact that their brains act more like "normal" peoples' when you give them drugs that augment these signaling pathways, or that we have a well-worked out body of theory that's fairly consistent with the evidence while this other stuff he lists has essentially none behind it. Surely not.
I've found 3x 1.6g doses of piracetam a day, each dose taken with 250mg centrophenoxine does an excellent job, along with 1g fish oil concentrate and 1.2g lecithin. Haven't noticed any negative body effects.
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This is by my doctor, who I recommend.
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And certainly nothing to do with all these dopamine/norepinephrine polymorphisms we keep finding they have, or the fact that their brains act more like "normal" peoples' when you give them drugs that augment these signaling pathways, or that we have a well-worked out body of theory that's fairly consistent with the evidence while this other stuff he lists has essentially none behind it. Surely not.
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